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mutations and trisomy 12 are linked to precise remodeling of chromatin activation and accessibility areas. More precisely, the epigenomic profile induced by MYD88
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gene in patients relapsing soon after treatment While using the BCL2 antagonist venetoclax. sixty six Resistance to those agents is connected to these mutations in all over 70% of instances, Whilst they are frequently subclonal as well as their unique function triggering resistance really should be confirmed.
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